Cilnidipine
has added lipophilicity arch to abiding anti-hypertensive aftereffect activated
with control of the bounden site. In 24-hour analytic assessment, once-daily
administering of cilnidipine produced claret burden abridgement for 24-hour
period. The inhibitory aftereffect on the N-type Ca2+ approach may accord an
added analytic advantage for the analysis of hypertension, such as abolishment
of reflex tachycardia.
As
catecholamines abet platelet activation via α2-receptor on the platelet
membrane, a abatement in nor-epinephrine levels by cilnidipine causes abrasion
of platelet activation.
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